Search results

Filters

  • Journals
  • Authors
  • Keywords
  • Date
  • Type

Search results

Number of results: 7
items per page: 25 50 75
Sort by:
Download PDF Download RIS Download Bibtex

Abstract

We define as preterm any newborn born before 37 weeks of gestation. The incidence of inguinal hernia is 1–4.4% among full term neonates and older children. In preterm newborns it is significantly more often, with an incidence that raises up to 30%. In this comprehensive review of the literature we provide evidence-based answers in various questions concerning the optimal treatment of inguinal hernias in preterm neonates. Such questions include the proper time of intervention, the choice of optimal anesthesia, the necessity for contralateral investigation in case of an ipsilateral hernia, the prevention of post-operative apnea and the choice between classic and laparoscopic surgical techniques.
Go to article

Bibliography

1. Lloyd D.A., Rintala R.: Inguinal hernia and hydrocele. In: O’Neill J.A., Rowe M.I., Grosfeld J.L., Fonkalsrud E.W., Coran A.G. (eds.) Pediatric surgery. Mosby, St Louis, 1998; pp. 1071–1086.
2. Lau S.T., Lee Y.H., Caty M.G.: Current management of hernias and hydroceles. Semin Pediatr Surg. 2007; 16: 50–57.
3. Vaos G., Gardikis S., Kambouri K., et al.: Optimal timing for repair of an inguinal hernia in premature infants. Pediatr Surg Int. 2010; 26: 379–385.
4. Crankson S.J., Al Tawil K., Al Namshan M., et al.: Management of inguinal hernia in premature infants: 10-year experience. J Indian Assoc Pediatr Surg. 2015; 20: 21–24.
5. Uemera S., Woodward A., Amenera R., et al.: Early repair of inguinal hernia in premature babies. Pediatr Surg Int. 1999; 15: 36–39.
6. Steward D.J.: Preterm infants are more prone to complications following minor surgery than are term infants. Anesthesiology. 1982; 56: 304–306.
7. Vogels H.D., Bruijnen C.J., Beasley S.W.: Predictors of recurrence after inguinal herniotomy in boys. Pediatr Surg Int. 2009; 25: 235–238.
8. Lautz T.B., Raval M.V., Reynolds M.: Does timing matter? A national perspective on the risk of incarceration in premature neonates with inguinal hernia. J Pediatr. 2011; 158: 573–577.
9. Antonoff M.B., Kreykes N.S., Saltsman D.A., et al.: American Academy of Pediatrics Section on Surgery hernia survey revisited. J Pediatr Surg. 2005; 40: 1009–1014.
10. Takahashi A., Toki F., Yamamoto H., et al.: Outcomes of herniotomy in premature infants: recent 10 year experience. Pediatr Int. 2012; 54: 491–495.
11. Lee S.L., Gleason J.M., Sydorak R.M.: A critical review of premature infants with inguinal hernias: optimal timing of repair, incarceration risk, and postoperative apnea. J Pediatr Surg. 2011; 46: 217–220.
12. Frumiento C., Abaijan J.: Spinal anesthesia for preterm infants undergoing inguinal hernia repair. Arch Surg. 2000; 135: 445–451.
13. Raveenthiran V.: Controversies Regarding Neonatal Inguinal Hernia. J Neonat Surg. 2014; 3: 31–34.
14. Esposito C., Turial S., Escolino M., et al.: Laparoscopic inguinal hernia repair in premature babies weighing 3 kg or less. Pediatr Surg Int. 2012; 28: 989–992.
15. Turial S., Enders J., Krause K., et al.: Laparoscopic inguinal herniorrhaphy in babies weighing 5 kg or less. Surg Endosc. 2011; 25: 72–78.
16. Chan I.H., Lau C.T., Chung P.H., et al.: Laparoscopic inguinal hernia repair in premature neonates: is it safe? Pediatr Surg Int. 2013; 29: 327–330.
17. Pastore V., Bartoli F.: Neonatal laparoscopic inguinal hernia repair a 3-year experience. Hernia. 2015; 19: 611–615.
18. Tackett L.D., Breur C.K., Luks F.I., et al.: Incidence of contralateral inguinal hernia: a prospective analysis. J Pediatr Surg. 1999; 34: 684–688.
19. Steven M., Greene O., Nelson A., et al.: Contralateral inguinal exploration in premature neonates: is it necessary? Pediatr Surg Int. 2010; 26: 703–706.
20. Marulaiah M., Atkinson J., Kukkady A., et al.: Is contralateral exploration necessary in preterm infants with unilateral inguinal hernia? J Pediatr Surg. 2006; 41:2004–2007. 21. Steigman C., Sotelo-Avila C., Weber T.: The incidence of spermatic cord structures in inguinal hernia sacs from male children. Am J Surg Pathol. 1999; 23: 880–885. 22. Dehner L.P.: Inguinal hernia in the male child: where the latest skirmish line has formed. Am J Surg Pathol. 1999; 23: 869–887. 23. Walc L., Bass J., Rubin S., et al.: Testicular fate after inguinal hernia repair and orchidopexy in patients under 6 months of age. J Pediatr Surg. 1995; 30: 1195–1197. 24. Laituri C.A., Garey C.L., Pieters B.J., et al.: Overnight observation in former premature infants undergoing inguinal hernia repair. J Pediatr Surg. 2012; 47: 217–220. 25. Walther-Larsen S., Rasmussen L.S.: The former preterm infant and riskof post-operative apnoea: recommendations for management. Acta Anesthsiol Scand. 2006; 50: 888–893. 26. Murphy J.J., Swanson T., Ansermino M., et al.: The frequency of apneas in premature infants after inguinal hernia repair: do they need overnight monitoring in the intensive care unit? J Pediatr Surg. 2008; 43: 865–868. 27. Özdemir T., Arıkan A.: Postoperative apnea after inguinal hernia repair in formerly premature infants: impacts of gestational age, postconceptional age and comorbidities. Pediatr Surg Int. 2013; 29: 801–804.
Go to article

Authors and Affiliations

Ioannis Patoulias
1
Ioanna Gkalonaki
1
ORCID: ORCID
Dimitrios Patoulias
2

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki, General Hospital “G Gennimatas”, Thessaloniki, Greece
  2. First Department of Internal Medicine, General Hospital “Hippokration”, Thessaloniki, Greece
Download PDF Download RIS Download Bibtex

Abstract

The combination of the functional disorders of urination and defecation constitutes the Dys-functional Elimination Syndrome (DES). DES refers to an abnormal pattern of elimination of unknown etiology characterized by bowel and bladder incontinence and withholding, with no underlying anatomic or neurologic abnormalities. Essential precondition for a child to be subsumed under this entity is the exclusion of either anatomical or neurological causative factors. In the present review study the individual entities of dysfunctional filling, such as the unstable or lazy bladder, or dysfunctional urination, such as the detrusor sphincter dyssynergia and the functional constipation are being described comprehensively. Subsequently, the analysis of the pathophysiological effects of the dysfunctional elimination syndrome such as incontinence, urinary tract infections and the conservation or the deterioration of vesicoureteric reflux, is being accentuated. With the documentation of DES, the therapeutic strategy should aim at treating both the functional disorder of the vesicourethral unit and the functional constipation. The first part does not specify depending on the type of this disorder. Rarely, surgical treatment of functional urinary disorders may be required.
Go to article

Bibliography

1. Shaikh N., Hoberman A., Wise B., et al.: Dysfunctional elimination syndrome: is it related to urinary tract infection or vesicoureteral reflux diagnosed early in life? Pediatrics. 2003 Nov; 112 (5): 1134–1137.
2. Halachmi S., Farhat W.A.: Interactions of constipation, dysfunctional elimination syndrome, and vesicoureteral reflux. Adv Urol. 2008; 2008: 828275.
3. Aydoğdu O., Burgu B., Teber S., et al.: A challenging review of childhood incontinence: rare complications of dysfunctional elimination syndrome in an epileptic boy. Turk J Pediatr. 2011 Jan– Feb; 53 (1): 100–103.
4. Von Gontard A., Hollmann E.: Comorbidity of functional urinary incontinence and encopresis: somatic and behavioral associations. J Urol. 2004 Jun; 171 (6 Pt 2): 2644–2647.
5. Curran M.J., Kaefer M., Peters C., Logigian E., Bauer S.B.: The overactive bladder in childhood: long- term results with conservative management. J Urol. 2000 Feb; 163 (2): 574–577.
6. Hadjizadeh N., Motamed F., Abdollahzade S., Rafiei S.: Association of voiding dysfunction with functional constipation. Indian Pediatr. 2009 Dec; 46 (12): 1093–1095. Epub 2009 Apr 1.
7. Klijn A.J., Asselman M., Vijverberg M.A., et al.: The diameter of the rectum on ultrasonography as a diagnostic tool for constipation in children with dysfunctional voiding. J Urol. 2004 Nov; 172 (5 Pt 1): 1986–1988.
8. Wein A.J., Kavoussi L.R., Campbell M.F.: Urology Cambell-Walsh, 10th ed. Saunders Elsevier: 2012; 3418–3420.
9. O’Regan S., Yazbeck S.: Constipation: a cause of enuresis, urinary tract infection and vesico-ureteral reflux in children. Med Hypotheses. 1985 Aug; 17 (4): 409–413.
10. O’Regan S., Yazbeck S., Schick E.: Constipation, bladder instability, urinary tract infection syndrome. Clin Nephrol. 1985 Mar; 23 (3): 152–154.
11. Ab E., Schoemaker M., Van Empelen R.: Paradoxical movement of the pelvic floor in dysfunctional voiding and the results of biofeedback training. Br J Urol Int. 2002; 89: 48.
12. Patoulias I.: Voiding disturbance in childhood. 1st ed. Parisianos, Athens: 2011; 58– 59. ISBN 978- 960-394-723-3.
13. Loening-Baucke V.: Urinary incontinence and urinary tract infection and their resolution with treatment of chronic constipation of childhood. Pediatrics. 1997 Aug; 100 (2 Pt 1): 228–232.
14. Chase J., Austin P., Hoebeke P., McKenna P.: International Children's Continence Society. The management of dysfunctional voiding in children: a report from the Standardisation Committee of the International Children’s Continence Society. J Urol. 2010 Apr; 183 (4): 1296–1302.
15. Hoebeke P., Van Laecke E., Van Camp C., Raes A., Van De Walle J.: One thousand video-urodynamic studies in children with non-neurogenic bladder sphincter dysfunction. BJU Int. 2001 Apr; 87 (6): 575–580.
16. Herndon C.D., Decambre M., McKenna P.H.: Interactive computer games for treatment of pelvic floor dysfunction. J Urol. 2001 Nov; 166 (5): 1893–1898.
17. Hansson S., Hjalmas K., Jodal U., Sixt R.: Lower urinary tract dysfunction in girls with untreated asymptomatic or cover bacteriuria. J Urol. 1990; 143: 333–336.
18. Issenman R.M., Filmer R.B., Gorski P.A.: A review of bowel and bladder control development in children: how gastrointestinal and urologic conditions relate to problems in toilet training. Pediatrics 1999; 103: 1346–1352.
19. Regan S.O., Schick E., Hamburger B., Yazbeck S.: Constipation associated with vesicoureteral reflux. Urol. 1986; 28: 394–396.
20. Chen J.J., Mao W., Homayoon K., Steinhardt G.F.: A multivariate analysis of dysfunction elimination syndrome, and its relationships with gender, urinary tract infection and vesicoureteral reflux in children. J Urol. 2004; 171: 1907–1910.
21. Naseer S.R., Steinhardt G.F.: New renal scars in children with urinary tract infections, vesicoureteral reflux and voiding dysfunction: a prospective evaluation. J Urol. 1997 Aug; 158 (2): 566–568.
22. Mulders M.M., Cobussen-Boekhorst H., de Gier R.P., Feitz W.F., Kortmann B.B.: Urotherapy in children: quantitative measurements of daytime urinary incontinence before and after treatment according to the new definitions of the International Children’s Continence Society. J Pediatr Urol. 2011 Apr; 7 (2): 213–218.
23. Nevéus T., Von Gontard A., Hoebeke P., et al.: The standardization of terminology of lower urinary tract function in children and adolescents: report from the Standardisation Committee of the International Children’s Continence Society. J Urol. 2006 Jul; 176 (1): 314–324.
24. Farhat W., Bägli D.J., Capolicchio G., et al.: The dysfunctional voiding scoring system: quantitative standardization of dysfunctional voiding symptoms in children. J Urol. 2000 Sep; 164 (3 Pt 2): 1011–1015.
25. Bower W.F., Yip S.K., Yeung C.K.: Dysfunctional elimination symptoms in childhood and adulthood. J Urol. 2005 Oct; 174 (4 Pt 2): 1623–1627; discussion 1627–1628.
26. Vereecken R.L., Proesmans W.: Urethral instability as an important element of dysfunctional voiding. J Urol. 2000; 163: 585–588.
27. Dede O., Sakellaris G.: Daytime urinary incontinence. Essentials in Pediatr Urol. 2012; 57–68.
28. Desantis D.J., Leonard M.P., Preston M.A., Barrowman N.J., Guerra L.A.: Effectiveness of biofeedback for dysfunctional elimination syndrome in pediatrics: a systematic review. J Pediatr Urol. 2011 Jun; 7 (3): 342–348.
29. Dyer L.L., Franco I.: Botulinum Toxin-A Therapy in pediatric Urology: Indications for the Neurogenic and Non-Neurogenic Neurogenic Bladder. Scientific World J. 2009; 9: 1300–1305.
30. Kroll P., Jankowski A., Soltysiak J., et al.: Botulinum toxin-A injections in children with neurogenic bladder. Nephroourol. 2011; 3: 125–128.
31. Carr L.K.: Botulinum toxin A should not be first-line therapy for overactive bladder. Can Urol Assoc J. 2011 Jun; 5 (3): 204–205.
32. Steele S.S.: Botulinum toxin A: First-line therapy for idiopathic detrusor over activity. Can Urol Assoc J. 2011; 5: 207–209.
33. Barroso U. Jr, Tourinho R., Lordêlo P., Hoebeke P., Chase J.: Electrical stimulation for lower urinary tract dysfunction in children: a systematic review of the literature. Neurourol Urodyn. 2011 Nov; 30 (8): 1429–1436.
34. Lordêlo P., Soares P.V., Maciel I., Macedo A. Jr, Barroso U. Jr.: Prospective study of transcutaneous parasacral electrical stimulation for overactive bladder in children: long-term results. J Urol. 2009 Dec; 182 (6): 2900–2904.
Go to article

Authors and Affiliations

Ioanna Gkalonaki
1
ORCID: ORCID
Ioannis Patoulias
1

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki Greece, General Hospital “G.Gennimatas”, Thessaloniki, Greece
Download PDF Download RIS Download Bibtex

Abstract

The main target during management of a male pediatric patient with clinical signs of acute scrotum is the timely diagnosis, in order not to jeopardize the viability of the affected testicle. Thorough evaluation of the patient’s medical history, symptomatology, clinical and ultrasonographic findings, con-stitutes the basis of the diagnostic procedure. After comprehensive research of the relevant literature, we highlight the remaining difficulties in the evaluation of the clinical and ultrasonographic findings for the accurate diagnosis of the acute scrotum. In conclusion, it is worth emphasizing on the following: a. the most common diseases that come under the diagnosis of the acute scrotum may present with similar symptoms, b. in neglected cases the diagnostic approach becomes more difficult, constituting the evalua-tion of the pathognomonic clinical signs challenging, and c. inability to exclude the diagnosis of spermatic cord torsion should be an indication for the surgical exploration of the affected hemiscrotum.
Go to article

Authors and Affiliations

Ioanna Gkalonaki
1
ORCID: ORCID
Ioannis Patoulias
1
Michail Anastasakis
1
Christina Panteli
1
Dimitrios Patoulias
2

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki, General Hospital “G. Gennimatas”, Thessaloniki, Greece
  2. First Department of Internal Medicine, General Hospital “Hippokration”, Thessaloniki, Greece
Download PDF Download RIS Download Bibtex

Abstract

The extremely rare localization of an intramuscular hemangioma (IMH) into the anterior scalene muscle was the motive for the present case report, aiming to highlight major, atypical characteristics. An 11-month-old boy with free medical history presented with a painless and progressively growing lesion 4.5 × 4 cm in diameter, located in the left supraclavicular region over the last 4 months. During physical examination, the presence of a painless, non-pulsating, non-adhesive to the overlying skin lesion was documented. Color Doppler flow ultrasonographic examination demonstrated the increased blood supply to the aforementioned lesion. Thus, we planned an elective surgical excision of the lesion in healthy limits. The postoperative course was uneventful, and the patient was discharged on the second postoperative day in good general condition. Histopathologic examination revealed the presence of hemangioma surrounded by connective tissue bundles and striated muscle fibers. IMHs do not follow the general rule of regression, beyond the age of 6–12 months, with no trend to increase over time. Accurate preoperative diagnosis is challenging. Color Doppler flow ultrasonographic examination is the imaging modality of choice during the preoperative assessment. Surgical excision of the IMH in healthy limits is the most appropriate treatment option.
Go to article

Authors and Affiliations

Ioannis Patoulias
1
Ioanna Gkalonaki
1
ORCID: ORCID
Magdalini Mitroudi
1
Thomas Feidantsis
1
Constantine Theocharidis
2
Dimitrios Patoulias
3

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki, General Hospital “G. Gennimatas”, Greece
  2. Department of Pathology, General Hospital “G. Gennimatas”, Thessaloniki, Greece
  3. Second Propedeutic Department of Internal Medicine, Aristotle University of Thessaloniki, General Hospital “Hippokration”, Greece
Download PDF Download RIS Download Bibtex

Abstract

Scar development in the children’s renal cortex with vesicoureteral reflux (VUR) is one of the most important parameters of prognosis. It can develop regardless of the chosen treatment, even after the regression of VUR. The shape of the renal papillae, the ascending urinary tract infection, the greater than third-degree VUR, and finally the increased intra-calyceal pressure, induce the formation of renal scarring in the renal parenchyma. Renal scarring may complicate VUR independently of the therapeutic strategy (conservative or operative) and its regression. For restitution of this entity, many scientific terms have been used and the most common of them is intrarenal reflux (IRR). The effects of VUR on future renal function result from the limited ability of the affected kidney to grow (failure of renal growth) due to the existence of scars in the renal cortex, the worsening of these scars, or finally the creation of new scars. With the present study, we intend to clarify the etiology and the pathophysiology of IRR and the relation of VUR prognosis to newer biomarkers such as N-acetyl-beta-glycosaminidase, beta-2 microglobulin, Pen-traxin- 3 and Liver-type fatty-acid-binding protein.
Go to article

Authors and Affiliations

Ioanna Gkalonaki
1
ORCID: ORCID
Evangelia Schoina
1
Michail Anastasakis
1
Ioannis Patoulias
1

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki, General Hospital “G. Gennimatas”, Thessaloniki, Greece
Download PDF Download RIS Download Bibtex

Abstract

Balanitis Xerotica Obliterans is a chronic, progressive, sclerosing inflammation of unclear etiology. It involves the external genitalia of males and more specifically the prepuce and its frenulum, the glans, and the external urethral meatus while it may extend to the peripheral part of the urethra. Recent studies have noted an increasing incidence in the paediatric population. It is the most common cause of secondary (pathologic) phimosis. Even more, in boys with physiologic phimosis that does not respond to conservative treatment, Balanitis Xerotica Obliterans should be considered as the underlying condition. In this study, we present all the latest data and attempt to create a diagnostic and curative algorithm regarding this condition.
Go to article

Bibliography

1. Hartley A., Ramanathan C., Siddiqui H.: The surgical treatment of Balanitis Xerotica Obliterans. Indian J Plast Surg. 2011 Jan; 44 (1): 91–97.
2. Depasquale I., Park A.J., Bracka A.: The treatment of balanitis xerotica obliterans. BJU International. 2000; 86 (4): 459–465.
3. Singh I., Ansari M.S.: Extensive Balanitis Xerotica Obliterans (BXO) involving the anterior urethra and scrotum. Int Urol Nephrol. 2006; 38: 505–506.
4. Hallopeau H.: Leçons cliniques sur les maladies cutanées et syphiliques. Union Med Can. 1887; 43: 472.
5. Depasquale I., Park A.J., Bracka A.: The treatment of Balanitis Xerotica obliterans. BJU Int 2000; 86: 459–465.
6. Catterall R.D., Oates J.K.: Treatment of Balanitis Xerotica obliterans with hydrocortisone injections. Br J Vener Dis. 1962; 38: 75–77.
7. Becker K.: Lichen Sclerosus in Boys. Dtsch Arztebl Int. 2011; 108 (4): 53–58.
8. Kizer W.S., Prarie T., Morey A.F.: Balanitis xerotica obliterans: Epidemiologic distribution in an equal access health care system. South Med J. 2003; 96: 9–11.
9. Jayakumar S., Antao B., Bevington O., Furness P., Ninan G.K.: Balanitis Xerotica obliterans in children and its incidence under the age of 5 years. J Ped Urol 2012; 8: 272–275.
10. Bochove-Overgaauw D., Gelders W., De Vyider A.: Rutine biopsies in pediatric circumcision (non) sense? J Pediatr Urol. 2009; 5: 178–180.
11. Pilatz A., Altinkilic B., Schormann E., et al.: Congenital phimosis in patients with and without lichen sclerosus: distinct expression patterns of tissue remodeling associated genes. J Uro. 2013; 189: 268e74.
12. Kizer W.S., Prarie T., Morey A.F.: Balanitis Xerotica Obliterans: epidemiologic distribution in an equal access health care system. South Med J. 2003; 96: 9e11.
13. Ebert A.K., Rösch W.H., Vogt T.: Safety and tolerability of adjuvant topical tacrolimus treatment in boys with lichen sclerosus: a prospective phase 2 study. Eur Urol. 2008; 54: 932e7.
14. Gargollo P., Kozakewich H., Bauer S., et al.: Balanitis Xerotica obliterans in boys. J Urol 2005; 174: 1409e12.
15. Kiss A., Csontai A., Pirot L., Nyirady P., Merksz M., Kiraly L.: The response of Balanitis Xerotica obliterans to local, steroid application compared with placebo in children. J Urol 2001; 165: 219e20.
16. Powell J., Wojnarowska F.: Lichen sclerosus. 1999; 353: 1777e89.
17. Peterson A.C., Palmineteri E., Lazzeri M., Guanzoni G., Barbagli G., Webster G.: Heroic measures may not always be justified in extensive urethral stricture due to lichen sclerosus (Balanitis Xerotica obliterans). J Urol. 2004; 64: 565–568.
18. Dillon W.I., Saeed G.M., Fivenson D.P.: Borrelia burgdorferi DNA is undetectable by polymerase chain reaction in skin lesions of morphoea, scleroderma or lichen sclerosus et atrophicus inpatients of North America. J Am AcadDermatol. 1995; 33: 617e20.
19. Pugliese J., Morey A., Peterson A.: Lichen sclerosus: review of the literature and current recommendations for management. J Urol. 2007; 178: 2268e76.
20. Meffert J.J., Davis B.M., Grimwood R.E.: Lichen sclerosus. J Am Acad Dermatol. 1995; 32: 393–416.
21. Powell J., Wojnarowska F.: Childhood vuvlvar lichen sclerosus: an increasingly common problem. J Am Acad Dermatol. 2001; 44: 803–806.
22. Depasquale I., Park A.J., Singh I., Ansari M.S.: Extensive Balanitis Xerotica Obliterans (BXO) involving the anterior urethra and scrotum. In Urol Nephrol. 2006; 38: 505–506.
23. Yardiey I.E., Cosgrove C., Lambert A.W.: Pediatric preputial pathology: are we circumcising enough? Ann R Coll Surg Engl. 2007; 89: 62–65.
24. Edmonds E., Barton G., Buisson S., et al.: Gene expression profiling in male genital lichen sclerosus. Int J ExpPathol. 2011; 92: 320e5.
25. Bale P.M., Lochhead A., Martin H.C., Gollow I.: Balanitis xerotica obliteransin children. Pediatr Pathol. 1987; 7: 617–627.
26. Mattioli G., Repetto P., Carlini C., Granata C., Gambini C., Jasonni V.: Lichen sclerosus et atrophicus in children with phimosis and hypospadias. Pediatr Surg Int. 2002; 18: 273e5.
27. Batbagli G., Palminteri E., Balo S., et al.: Lichen sclerosus of the male genitalia and urethral stricture diseases. Urol Int. 2004; 73: 1–5.
28. Celis S., Reed F., Murphy F., et al.: Balanitis xerotica obliteransin in children and adolescents: A literature review and clinical series. J Pediatr Urol. 2014; 10: 34–39.
29. Kiss A., Király L., Kutasy B., Merksz M.: High incidence of Balanitis Xerotica Obliterans in boys with phimosis: prospective 10-year study. Ped Dermatol. 2005; 22: 305–308.
30. Bale P.M., Lochhead A., Martin H.C., et al.: BXO in children Pediatr Pathol. 1987; 7: 617; 9.
31. Mohammed A., Shegil I.S., Christou D., Khan A., Barua J.M.: Paediatric Balanitis Xerotica obliterans: an 8-year experience. Arch Ital Urol Androl. 2012; 84: 12e6.
32. Vincent M., MacKinnon E.: The response of clinical Balanitis Xerotica Obliterans to the application of topical steroid-based creams. J Pediatr Surg. 2005; 40: 709e12.
33. Holbrook C., Tsang T.: Management of boys with abnormal appearance of meatus at circumcision for BXO. Ann R Coll Surg Engl. 2011; 93: 482–484.
34. Barbagli G., Palminteri E., Baló S., et al.: Lichen sclerosus of the male genitalia and urethral stricture deseases. Urol Int. 2004; 73: 1–5.
35. Pugliese J., Morey A., Peterson A.: Lichen sclerosus: review ofthe literature and current recommendations for management. J Urol. 2007; 178: 2268e76.
36. Nasca M.R., Innocenzi D., Micali G.: Penile cancer among patients with genital lichen sclerosus. J Am Acad Dermatol. 1999; 41: 911e4.
37. Pietrzak P., Hadway P., Corbishley C., Watkin N.: Is the association between Balanitis Xerotica Obliterans and penile carcinoma underestimated? BJU Int. 2006; 98: 74e6.
38. Prowse D.M., Ktori E.N., Chandrasekaran D., et al.: Human papillomavirus-associated increase in p16-INK4A expression in penile lichen sclerosus and squamous cell carcinoma. Br J Dermatol. 2008; 158: 261–265.
39. Neill S.M., Tatnall F.M., Cox N.H.: Guidelines for the management of lichen sclerosus. Br J Dermatol. 2002; 147: 640–649.
40. Poindexter G., Morrell D.S.: Anogenital pruritus: lichen sclerosus in children. Pediatr Ann. 2007; 36: 785–791.
Go to article

Authors and Affiliations

Ioanna Gkalonaki
1
ORCID: ORCID
Michalis Anastasakis
1
Ioanna Sofia Psarrakou
2
Ioannis Patoulias
1

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki Greece, General Hospital “G.Gennimatas”, Thessaloniki, Greece
  2. Department of Pediatrics, General Hospital “G. Gennimatas”, Thessaloniki, Greece
Download PDF Download RIS Download Bibtex

Abstract

Hutch Diverticulum (HD) is defined as the protrusion of the mucosal and submucosal layer through the muscle bundles of the underlying detrusor muscle. HD is located at the vesicoureteral junction with a backward direction from the homolateral ureteral orifice. As far as its etiology is con-cerned, HD is caused either by a congenital muscle wall defect at the level where the Waldeyer’s fascia occupies the clefts between the vesical part of the homolateral ureter and the detrusor, or is associated with abortive ureteral duplication or defective incorporation of mesonephric duct into the bladder at the site of ureteral hiatus or finally is associated with the development of transient urethral obstruction. HD is usually unilateral and more common in male patients. It may be associated with the Ehlers-Danlos, Williams-Elfin and Menkes syndromes. HD usually occurs in childhood and rarely during adulthood. It is found in 0.2–13% of all children presenting with urinary tract infection. Through this short review article, we attempt to present in detail the most recent bibliographic data concerning this entity, focusing on pathophysiology, diagnostic approach, and treatment strategy.
Go to article

Authors and Affiliations

Ioanna Gkalonaki
1
ORCID: ORCID
Michail Anastasakis
1
Christina Panteli
1
Ioannis Patoulias
1

  1. First Department of Pediatric Surgery, Aristotle University of Thessaloniki, General Hospital “G. Gennimatas”, Thessaloniki, Greece

This page uses 'cookies'. Learn more